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Top 100 AIDS Science Inconsistencies
Top 100 AIDS Science Inconsistencies

by John Kirkham 11th January 2003.

All the inconsistencies below can be substantiated by independent research. The persistence of the HIV=AIDS=Death dogma is truly astonishing, in the face of so many specific scientific flaws.

1 A new medical definition (Idiopathic CD4+ T-cell lymphocytopenia) was created to avoid the fact that AIDS occurs in the absence of HIV (65, 87)

2 HIV does not satisfy Koch's postulates, the criteria that must be met in order to prove that a microbe causes a disease (90)

3 Anti-HIV drugs, including protease inhibitors, destroy T-cells (4 - 10)

4 Septrin (also called Septra, Bactrim, Co-trimoxazole) and anti-HIV drugs destroy mitochondria (11, 12)

5 The PCP (Pneumocystis Carinii pneumonia) fungus becomes resistant to Septrin (12)

6 Recreational drugs (heroin, poppers, crystal met, ecstasy, cocaine) reduce CD4 cell numbers (13 - 18, 58, 66 - 68)

7 HIV positive patients recover after they stop taking drugs (58)

8 Recreational drugs cause AIDS-defining diseases (see table 7 of 58)

9 Anti-HIV drugs cause AIDS-defining diseases (58)

10 Anti-HIV drugs inhibit human enzymes (11)

11 HIV positive Africans in dire poverty in Uganda and no access to anti-HIV drugs lived as long as HIV positives in the west who took anti-HIV drugs (33)

12 There are no comparative studies of survival in HIV negatives and combo-free HIV positive heterosexuals with no other risk factors

13 Only 38% of healthy long-term positives had ever used AZT or other nuleoside analogs Compared with 94% of progressors (80)

14 Decreases in AIDS cases preceded the introduction of new drug treatments (Dec 1995) by three full years (see fig.6 of 106)

15 Anti-HIV drugs have anti-microbial effects (49, 50, 10)

16 The introduction of AZT (1987) did not cause a decline in the AIDS death rate (105)

17 In the only long term trial of AZT (The Concorde study) 172 participants died, 169 while taking AZT, 3 while on placebo (51)

18 Nucleoside analog drugs suppress/destroy the bone marrow where all immune system cells are born (26, 32, 111)

19 HIV+ children born to AZT treated mothers had a higher probability of developing severe disease or severe immunsuppression (53)

20 "Drug holidays" recover immune responses

21 AZT caused the same transient increase in CD4 count in HIV negatives as in HIV positives (55)

22 There are no controlled studies showing that AIDS occurs in the absence of all other possible non-HIV causal factors

23 Long-living, healthy, drug-free HIV positives are mostly ignored by AIDS researchers

24 Apart from the early (fraudulent) AZT studies and the Concorde study no efficacy studies compare drugs with placebo

25 There are well documented, non-HIV causes for every AIDS disease

26 The incidence of AIDS-defining diseases among Western non-drug users has not been shown to exceed national backgrounds (58)

27 Early AIDS coincided with the cumulative effects of unprecedented, intense use of volatile nitrite (poppers) as an aphrodisiac marketed almost exclusively to homosexuals (102)

28 AIDS can be treated effectively without anti-HIV drugs (39 - 42)

29 On average viral load overestimates infectious HIV by a factor of 60,000 (21)

30 Even a PCR method that can detect 1 infected cell in 100000 found very little HIV DNA in HIV positives (23)

31 HIV could not be cultured from people with a detectable viral load (19, 21)

32 HIV has never been properly isolated (20)

33 After many billions of dollars of research effort over 20 years, HIV scientists still cannot explain how HIV causes AIDS

34 After many billions of dollars of research effort over 20 years there is no vaccine and no cure, there are only toxic drugs

35 There was no increase in HIV seroprevalence outside risk groups in the UK despite record STD rates and teenage pregnancy rates (25)

36 HIV DNA was found to be constant from the time of seroconversion, but CD4 count continually went down (29)

37 CD4 count goes down and viral load goes up while on the anti-HIV drugs

38 AZT is hardly triphosphorylated by the body so it cannot possibly have an anti-HIV effect (30)

39 AZT has no effect on HIV DNA but makes viral load (HIV RNA) go down (31)

40 Research throughout the 1970s showed that retroviruses do not kill cells

41 The probability of heterosexual transmission of HIV was found to be very low (1 in a 1000) (34)

42 HIV antibody tests give repeated false positives and seroreversions can occur

43 HIV tests are sensitive to nonspecific antibody binding

44 HIV tests involve an arbitrary dilution factor, everyone tests positive (because of nonspecific antibody binding) if their serum is undiluted (104)

45 All the proteins used in the HIV test are associated with retroviral genes that are found naturally (endogenous) in all humans (72)

46 Endogenous retroviruses can generate immune responses in humans (73, 74)

47 None of the HIV proteins tested for have been proven to belong to HIV (75)

48 There are over 60 different conditions, including pregnancy, that have been known to generate false positives on the HIV test (91)

49 The Elisa, Western Blot and PCR tests for HIV all carry disclaimers nullifying their detection of HIV

50 The criteria for HIV-positivity used in the antibody tests varies between countries and between organizations within a country and can produce indeterminate (neither positive or negative) results (75, 109)

51 The viral load PCR primers were found to be nonspecific for "HIV" genetic sequences (35)

52 The viral load test gives false negatives (36)

53 The viral load test gives false positives (36)

54 The viral load test has low reproducibility (36 - 38)

55 Direct measurements showed no correlation between viral load and CD4 count (43)

56 Many conditions cause reduced CD4 counts (86)

57 CD4 counts between 200 and 300 have been observed in healthy HIV negatives (87)

58 There are no studies comparing CD4 cell variations in combo-free HIV positives (with no risk factors) and HIV negatives

59 According to the AIDS establishment, a heterosexual AIDS "epidemic" of African origin started off in the West as a homosexual "epidemic"

60 In 1985 HIV incidence in Southern Africa was confined to homosexuals who had been to the US and those who had had sex with them (88, 89)

61 The USA was found to be the world's most sexually promiscuous nation (27)

62 Condoms (made from polyisoprene) have holes in much larger than HIV (28, 110)

63 Reducing STD incidence in Africa did not reduce the rate of HIV seroconversion * (101)

64 Only a minute proportion of Africans have actually been tested for HIV, seroprevalence estimates are derived from extrapolations based on unrepresentative samples from maternity clinics

65 In Africa a single positive ELISA test or even a single "rapid" (saliva/urine) test is considered proof of HIV infection, "proof" in the developed world requires a series of tests

66 HIV seroprevalence was found to be much lower in South African prisons than in the general population (1)

67 The vast majority of African "AIDS patients" tested HIV negative (44, 45)

68 In "AIDS ravaged" Zambia since 1980 the population has increased and even the rate of increase in population has increased! (46)

69 In "AIDS ravaged" South Africa many coffin makers are either doing a slack trade or have gone out of business (47)

70 The total number of AIDS cases in Africa consists almost entirely of estimated cases rather than known, registered cases (54)

71 PCP is the typical AIDS defining disease in Western adults but it is almost entirely confined to young children in Africa (2, 3)

72 There is no Western heterosexual AIDS epidemic

73 IVDUs who consistently used a clean needle exchange program were 10.2 to 22.9 times MORE likely to test HIV positive than nonusers (48)

74 Non-human primates "progress" to AIDS (SAIDS) much quicker than humans do (107)

75 SIV does not cause SAIDS in wild primate populations (108)

76 SIV seroprevalence is too low in wild primate populations to account for SIV resistance in these populations (22)

77 SIV seroprevalence in captive SIV naïve primate populations was found to be very low (22)

78 Until the early 1930s many thousands of European men received transplants from chimpanzees and did not get AIDS (62)

79 Uganda study showed HIV-positivity did not indicate a new cause of disease, only decreased mortality in HIV negatives (52)

80 One thousand medical staff a year accidentally contract hepatitis from needles yet by 1998 there were no documented cases of surgeons or emergency medical technicians/paramedics getting AIDS, or even HIV, from occupational exposure (58, Table 16 of 106 )

81 All AIDS patients have lowered levels of glutathione, the major water soluble intracellular antioxidant (59, 60)

82 The antioxidant N-acetyl cysteine inhibits "HIV replication" (61)

83 Reactive oxygen species are implicated in the induction of HIV expression and cell death (40)

84 Treatment with oxidizing, mitogenic*** agents is necessary for HIV "isolation" from cell culture (56, 57)

85 Rectally deposited sperm can be immunosuppressive, mitogenic, oxidizing and a stimulator of antigen production (94 -100)

86 Low T-cell counts were shown to occur before HIV seroconversion and to predict seroconversion (92, 93)

87 HIV-like genetic sequences have been found in the HIV negative human genome (63)

88 Epitopes** of HIV regulatory proteins tat, rev and nef are expressed in normal human tissue (71)

89 Toxic intracellular stresses can create novel genetic sequences (64)

90 HIV showed over 40% variation in an essential gene (protease) sequence (103)

91 Foreign protein transfusions were found to be immune suppressive (79, 81, 84, 85)

92 Hemophiliacs can have hypergammaglobulinaemia which can cause false HIV positive test results (69)

93 Up to 99.9% of HIV genomes in plasma may be defective (70)

94 Mortality in hemophiliacs began to increase in exactly the same year they began taking AZT (81, 82)

95 The AIDS risk of hemophiliacs on AZT was 4.5 times higher, and mortality 2.4 times higher, than untreated controls (83)

96 Infectious HIV (a delicate virus) does not survive the Factor VIII preparation process (76 - 78)

97 HIV theorists have made incorrect predictions throughout the HIV era

98 Corticosteroids and endogenous cortisol suppress cellular immune responses and cortisol destroys immature T-cells (24)

99 Effective cellular immunity relies upon nitric oxide gas defense, see for example Eur. J. Immunol. 2002, 32(5):1455-63 100 AIDS spreads non-exponentially, unlike infectious disease (58)These inconsistencies have been researched and compiled by John Kirkham, who has a Master of Research Degree in Science and has been peer-review published. Table and HTML presentation by Fintan Dunne, Editor,


* Conversion from HIV negative to HIV positive

** Epitopes are parts of a molecule against which antibodies are made

*** Stimulates cell division



(2) American J of Respiratory and Critical Care Medicine 1994, 149(6):1591-1596

(3) Central African J of Medicine 1999, 45:127-8

(4) J Virol 2002, 76(12):5966-73 (5) J Biol Chem 1989, 264:6127-33

(6) Antimicrobial Agents and Chemotherapy 1990, 34:637-641

(7) Antiviral Chemistry and Chemotherapy 1991, 2:125-132

(8) AIDS 1989, 3:417-422

(9) NEJM 1987, 317:192-197

(10) Physicians Desk Reference 1999

(11) Nature Medicine 1995, 1(5):417-422


(13) Pharmacotherapy 1984, 4:284-291

(14) Cancer Research 1983, 43:1365-1371

(15) Lancet 1982, Feb 20, 412-416

(16) AIDS 1991, 5:35-41

(17) Annals NY Acad. Sci. 1987, 496:711-21

(18) Life Sciences 2001, 69:2931-2941

(19) NEJM 1995, 332:201-208

(20) Virol. 1997, 230:125-133

(21) Science 1993, 259:1749-1754


(23) J Virol 1990, 64: 864-872

(24) Medical Hypothesis 1996, 46:551-555

(25) The Times (UK) June 2nd 2001.

(26) Adverse Drug Reaction Bulletin 1996, 178:675-8

(27) Durex Global Sex Survey 2001, see also

(28) Rubber Chemistry and Technology, 1989, 62(4):683-697 (see page 692)

(29) J. AIDS 1994, 7:381-388

(30) Current Medical Research and Opinion 1999, Vol. 15, supplement 1

(31) J. AIDS 1991, 4:766-9

(32) Manufacturers insert at ( Bone Marrow Suppression)


(34) American J. Epidemiology 1997, 146(4):350-357

(35) AIDS 1998, 12:2076-2077

(36) Annals of Internal Medicine 1996, 124:803-815

(37) J. of AIDS and Human Retrovirology 1997, 15(2):174-5

(38) J. AIDS 1992, 5(9):872-877

(39) Proc. Nat. Acad. Sci. USA 1997, 94:1967-1972

(40) Medical Hypothesis 1993, 40(2):85-92

(41) Trans. Assoc. Am. Phys. 1984, 97:70-79

(42) Proc. Soc. Exp. Biol. Med. 1997, 216:201-210

(43) Nature Medicine 1999, 5(1):83-89 (see fig. 4b)

(44) J. AIDS 1994, 7(8):876-877

(45) Lancet 1992, 340:971-972



(48) American J. Epidemiology 1997, 146(12):994-1002 (see table 5)

(49) J. of Infectious Diseases 2000, 181:1629-1634

(50) J. of Infectious Diseases 1999, 180:448-453 (51) Lancet 1994, 343:871-881

(52) Lancet 1994, 343:1021-1023 (53) AIDS 1999, 13(8):927-33

(54) See tabulated data in the annex to the WHO Global Report 1998

(55) AIDS 1996, 10(12):1444-5

(56) Science 1986, 231:850-853 (57) Nature 1986, 319:10-11

(58) Genetica 1998, 104:85-132

(59) FASEB J. 1997, 11:1077-1089

(60) Proc. Natl. Acad. Sci. USA 1997, 94:1967-1972

(61) Proc. Natl. Acad. Sci. USA 1991, 88:986-990

(62) Hamilton D. The Monkey Gland Affair, Chatto and Windus Ltd., London 1986

(63) J. Virol. 1992, 66:2170-2179

(64) Clin. Diagn. Lab. Immunol. 1992, 6(3):330-335

(65) Biotechnology 1993, 11:955-956

(66) AIDS 1987, 1:105-111

(67) American J. Epidemiology 1993, 137(9):989-1000

(68) Clin. Immunol. Immunopathol. 1994, 70:245-250

(69) Isr. J. Med. Sci. 1991, 27:557-561

(70) Nature 1993, 364:291

(71) Am. J. Pathol. 1992, 141:1209-1216

(72) JAMA 1988, 260(5):674-679

(73) Immunological Reviews 1996, 152:193-236

(74) Proc. Natl. Acad. Sci. USA 1996, 93:5177-5184

(75) Biotechnology 1993, 11:696-707

(76) CDC Fact sheet on HIV transmission January, 1994

(77) JAMA 1989, 261:1275

(78) J. AIDS 1992, 5:822-828

(79) Ann. Int. Med. 1985, 103:723-726

(80) AIDS 1994, 8:1123 (81) Genetica 1995, 95:51-70

(82) Lancet 1995, 346:1371-1372

(83) Lancet 1994, 344:791-792, see table on page 791

(84) NEJM 1984, 322:941-949

(85) Am. J. Hematol. 1985, 20:1-6


(87) J. Antimicrobial Therapy 1996, 37(Suppl. B):171-83 (88) S. Afr. Med. J. 1985, 68(8):617-8

(89) NEJM 1985 312(19):1257-8.

(90) See "full text" at


(92) J. AIDS 1993, 6:820-822

(93) Epidemiology 1990, 1:453-459

(94) JAMA 1984, 251:237

(95) Br. Med. J. 1983, 286:1651

(96) NEJM 1983, 308:1181

(97) Fed. Proc. 1983, 42:1334

(98) J. Exp. Med. 1982, 155:1719

(99) Immunol. Today 1984, 5:357

(100) Theor. Biol. 1982, 96:741

(101) Lancet 1999, 353:525-535

(102) Kitzerow M. The AIDS Indictment, MRKCO Publishing, Chicago, 2000.

(103) Nature Medicine 1996, 4(7):753-759



(106) CDC HIV/AIDS Surveillance Report, Year-end 1997, (Vol.9 No.2)

(107) Science 1990, 248:1109-1112

(108) J. Virol. 2001, 75:2262-2275

(109) The Perth Group, Mother to Child Transmission of HIV and its Prevention with AZT and Nevirapine, ISBN 1876763728, page 5.




HIV=AIDS Controversy:
Test Ban The British publication, Continuum, has proposed a test ban on all HIV antibody tests for reasons given below.

We invite you to join... Continuum's Ban the HIV Test Campaign

The HIV antibody tests are unproved for HIV isolation because: There is no universal gold standard HIV isolate, therefore there can be no calculation of the specificity of any hypothetical HIV test. The only scientifically valid method of determining the specificity of antibodies present in sera of either healthy individuals or AIDS patients is the use of HIV isolation as a gold standard.

An antigen/antibody reaction cannot prove that the origin of certain proteins is a retrovirus or that antibodies are specifically directed against the retrovirus. The protein antigens that are used for the HIV tests are the biological outcome of stressed white blood cells used in the laboratory, and the antibodies that react with them can be generated by fungal and mycobacterial infections including PCP and Candida.

The HIV test has never been scientifically validated. HIV positive results are triggered by a wide variety of conditions and different test kits produce widely differing results. Factors known to cause False-Positive HIV Antibody Test Results: Flu vaccination, Leprosy, Tuberculosis, Renal (kidney) failure, Herpes simplex 1 & 2, Malaria, Hepatitis B vaccination, Organ transplantation, Epstein-Barr virus, Lymphoma, Autoimmune disease, Tetanus vaccination, MAI, Rheumatoid arthritis.

There is no scientific evidence of antibodies or proteins specific to HIV. There is no data proving the actual origin of either HIV proteins or antibodies, and there is ample evidence that reactions between HIV proteins and HIV antibodies are non-specific.

No particle of HIV has ever been obtained pure, free of contaminants; nor has a complete piece of HIV RNA (or transcribed DNA) ever been proved to exist. In the journal Virology (March 1997), Han Gelderblom, et al. disclosed that there were major contaminants in pure HIV which demonstrates that HIV does not exist as a unique, exogenous retrovirus but an excess of vesicles (particles of cellular proteins). What has been wrongly classified as HIV is an endogenous collection of gene products that can appear when immune system cells are chronically stimulated in the body or drastically manipulated in the laboratory.

Prejudice drives HIV test results: it has been confirmed that HIV antibody test labs use risk-factor information before deciding on results. If the individual falls into any of the 15 or more high-risk categories (including being gay, bi-sexual, prostitute or from a developing country)an indeterminate result may be read as HIV antibody positive.

There is no scientific basis for using these non-specific HIV tests to prove HIV infection or for making any antiviral health decisions based on them. We urge you to join our campaign to have the fraudulent HIV tests banned. Say NO to fraudulent HIV testing.

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Address: 172 Foundling Court,
Brunswick Centre London WC1N 1QE,
United Kingdom.

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